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Low Carb Sugar-Free Sweets & Cakes
Artinci was born out of Aarti's and Sumit's (Artinci's founders) abiding love for great-tasting dessert, while helping them stay committed to their health goals as well. As a result, Artinci makes delicious desserts with zero sugar, that are science and evidence-backed.
Aarti and Sumit come from a family of three generations of diabetics. They were themselves diagnosed pre-diabetic in 2012, and right there began a lifelong quest of a healthy, active lifestyle, including healthy swaps in food
Sugar free Sweets & Cakes
Aarti Laxman (Founder)
Artinci is founded by Aarti Laxman, a certified Metabolic coach in the Low-Carb Nutrition & Metabolic Health domain from dLife.in, India’s only legally tenable course in this subject—recognized by the NSDC (under the Ministry of Skill Development & Entrepreneurship, Govt. of India). It’s also internationally accredited by the CPD Standards Office UK, with a global record of 144 CPD hours—the highest for any course of its kind. The accreditation is both nationally valid and globally recognised in over 50+ countries..
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All about Sugar and sugar-free
The Science Behind Fibre and Insulin Sensitivity Plant-based diets, rich in natural fibers from fruits, vegetables, pulses, and whole grains, are strongly associated with lower diabetes risk. The primary reason extends beyond calorie control — it lies in how fiber interacts with the trillions of microbes in your gut. Dietary fibers, especially soluble and fermentable types (like in oats, legumes, banana, onions, and flaxseeds), escape digestion in the upper gut and reach the colon, where they become food for beneficial bacteria such as Bifidobacterium, Faecalibacterium prausnitzii, and Roseburia. These microbes ferment the fiber, producing short-chain fatty acids (SCFAs) — mainly acetate, propionate, and butyrate. SCFAs: The Microbial Messengers of Metabolic Health SCFAs act as signaling molecules that influence key aspects of metabolism and insulin sensitivity: Butyrate fuels the cells lining your intestine, maintaining strong gut-barrier integrity and preventing inflammation-driven insulin resistance. Propionate travels to the liver, helping regulate gluconeogenesis (glucose production). Acetate influences fat metabolism and appetite regulation through hormonal pathways such as GLP-1 and PYY. Together, SCFAs reduce chronic, low-grade inflammation — often called “metabolic inflammation” — that underlies insulin resistance. They also improve muscle and liver insulin signaling, making the body more responsive to insulin. Fiber-Fermenting Bacteria and Type 2 Diabetes Prevention Studies show that people who consume fiber-rich, plant-forward diets harbor greater microbial diversity and more fiber-fermenting bacteria compared to those eating low-fiber or animal-heavy diets. This microbial shift supports higher SCFA production and healthier glucose metabolism. Clinical research confirms that high-fiber diets can: Lower fasting blood glucose and HbA1c levels. Enhance gut microbial diversity. Increase SCFA production. Reduce inflammatory markers like CRP and endotoxins. These effects occur even without major calorie restriction, reinforcing the protective role of gut-derived metabolites like SCFAs. Building a SCFA-Boosting Diet To encourage SCFA production and insulin sensitivity naturally, focus on: Prebiotic fibers: From foods like garlic, onions, leeks, asparagus, bananas, and legumes. Whole grains: Oats, barley, and brown rice stabilize blood sugar and feed good microbes. Fermented foods: Dahi (yogurt), idli, dosa batter, and pickles enhance microbial resilience. Colorful plant diversity: Aim for 30+ plant varieties weekly — fiber types matter as much as quantity. Pair these foods with daily physical activity, adequate hydration, and sleep, all of which further improve metabolic balance and microbial health.
Can Healing Your Gut Lower Type 2 Diabetes Risk? Early Microbiome Shifts You Shouldn’t Ignore
How the Gut and Diabetes Are Linked Several large human studies now show that people with insulin resistance, prediabetes, or early type 2 diabetes have a distinct gut microbiome pattern compared with people with normal glucose levels. These patterns include reduced microbial diversity, shifts in specific bacterial groups, and altered production of metabolites that influence blood sugar and inflammation. Evidence from genetic and mechanistic studies suggests this is not just correlation: certain bacterial families and species appear to have a causal association with type 2 diabetes risk, either increasing or lowering the odds of developing the disease. Animal and human experiments also show that transferring an “unhealthy” microbiome can worsen glucose control, while correcting microbial imbalances can improve metabolic markers. Microbiome Shifts Seen Before Diabetes Onset Prospective and systematic studies indicate that gut changes can be detected in prediabetes and newly diagnosed, untreated type 2 diabetes, often years before full‑blown disease. People at this early stage commonly show a drop in beneficial butyrate‑producing bacteria, reduced overall diversity, and an increase in microbes linked with inflammation and impaired glucose metabolism. Some research using Mendelian randomization has identified specific taxa whose higher abundance is associated with increased type 2 diabetes risk, while others, such as some Lactobacillaceae and Bifidobacterium longum, are linked to lower risk. These early microbial signatures are being explored as potential biomarkers to identify individuals at high risk before fasting glucose or HbA1c become abnormal. Leaky Gut, Inflammation, and Insulin Resistance An unhealthy microbiome can weaken the intestinal barrier and increase gut permeability, often referred to as “leaky gut.” This allows bacterial components like lipopolysaccharide (LPS) to enter the bloodstream, where they activate immune receptors and trigger chronic low‑grade inflammation that interferes with insulin signaling. Studies in humans show that insulin resistance is associated with higher gut permeability even after accounting for obesity, suggesting that barrier dysfunction is part of the early diabetogenic process. Experimental work has also revealed specific microbial mechanisms—for example, reduced ability of the obese microbiota to metabolize ethanolamine can drive permeability, inflammation, and glucose abnormalities, which can be partly reversed with targeted probiotic strategies. Can Healing Your Gut Lower Diabetes Risk? Current evidence supports a model where improving gut health may help lower the risk of progressing from insulin resistance or prediabetes to type 2 diabetes, although definitive prevention trials are still ongoing. Observational and interventional data consistently show that diets rich in plant fiber, minimally processed foods, and healthy fats support a more diverse, metabolically favorable microbiome and better glucose control. Key gut‑supportive habits that may reduce type 2 diabetes risk include: Eating plenty of soluble and fermentable fiber from vegetables, fruits with peel, whole grains, legumes, nuts, and seeds to fuel beneficial short‑chain‑fatty‑acid‑producing bacteria. Including fermented foods (curd, yogurt, kefir, pickles without added sugar, traditional ferments) that can increase microbial diversity and support barrier integrity. Limiting ultra‑processed foods, refined carbohydrates, and excessive saturated fat, which are associated with dysbiosis, endotoxemia, and insulin resistance. Maintaining a healthy weight, moving regularly, sleeping well, and managing stress, all of which have measurable effects on the microbiome and insulin sensitivity. For people at high risk—such as those with family history, obesity, or prediabetes—combining gut‑friendly nutrition with standard medical advice (screening, weight management, physical activity, and when appropriate, medications) offers a practical, science‑aligned way to act early rather than waiting for diabetes to appear. https://gut.bmj.com/content/72/10/1848 https://pmc.ncbi.nlm.nih.gov/articles/PMC7457818/ https://www.frontiersin.org/journals/microbiology/articles/10.3389/fmicb.2024.1451054/full https://pmc.ncbi.nlm.nih.gov/articles/PMC9777768/
डायबिटिक के लिए गट-फ्रेंडली 7 आदतें: खाना, नींद, तनाव और व्यायाम
आदत 1: फाइबर से भरपूर देसी थाली अपनाएं हर भोजन में 25-30 ग्राम फाइबर लें – बाजरा, ज्वार रोटी, दालें, हरी सब्जियां। ये अच्छे बैक्टीरिया को खिलाते हैं, SCFAs बनाते हैं जो इंसुलिन रेसिस्टेंस कम करते हैं। सफेद चावल-मैदा छोड़ें। आदत 2: प्रोबायोटिक्स और प्रीबायोटिक्स का कॉम्बो दही, छाछ या घर का अचार रोज लें (प्रोबायोटिक्स), साथ में केला, प्याज (प्रीबायोटिक्स)। यह माइक्रोबायोम डाइवर्सिटी 20-30% बढ़ाता है, ब्लड शुगर स्थिर रखता है। आदत 3: 7-8 घंटे गहरी नींद लें रात 10 बजे सोएं, सुबह 6 बजे उठें। नींद की कमी से डिस्बायोसिस बढ़ता है, जो इंसुलिन सिग्नलिंग बिगाड़ता है। डिनर हल्का रखें। आदत 4: तनाव कम करने के लिए प्राणायाम रोज 10 मिनट अनुलोम-विलोम या भ्रामरी करें। कोर्टिसोल तनाव से leaky gut होता है, जो सूजन बढ़ाकर शुगर बिगाड़ता है। ध्यान से गट बैरियर मजबूत होता है। आदत 5: रोज 30 मिनट वॉक या योग सुबह टहलें या सूर्य नमस्कार करें। व्यायाम SCFAs उत्पादन बढ़ाता है, इंसुलिन संवेदनशीलता सुधारता है। पोस्ट-मील वॉक से शुगर स्पाइक 25% कम। आदत 6: पानी और हाइड्रेशन का ध्यान दिनभर 3-4 लीटर पानी पिएं, नारियल पानी या छाछ शामिल करें। डिहाइड्रेशन से कब्ज होता है, जो डिस्बायोसिस बढ़ाता है। आदत 7: अल्ट्रा-प्रोसेस्ड फूड अवॉइड करें पैकेट चिप्स, सोडा, रेडी-टू-ईट छोड़ें। ये बैड बैक्टीरिया को बढ़ावा देते हैं, endotoxins रिलीज करते हैं। घर का ताजा खाना चुनें। इन 7 आदतों को 21 दिन फॉलो करें – HbA1c में सुधार दिखेगा, गट हेल्थ बेहतर होगी। डॉक्टर से सलाह लें।
फाइबर, देसी खाना और गट माइक्रोबायोम: भारतीय थाली से शुगर कंट्रोल कैसे बेहतर हो सकता है?
गट माइक्रोबायोम क्या है और डायबिटीज से कैसे जुड़ा? आंतों में अरबों बैक्टीरिया का समूह गट माइक्रोबायोम कहलाता है, जो खाना पचाने, इम्यूनिटी और ब्लड शुगर रेगुलेशन में मदद करता है। असंतुलन (डिस्बायोसिस) से इंसुलिन रेसिस्टेंस बढ़ता है, जो टाइप-2 डायबिटीज का मुख्य कारण है। भारतीय आहार में फाइबर इस बैलेंस को बहाल करता है। फाइबर: अच्छे बैक्टीरिया का ईंधन फाइबर (जैसे सोलuble और insoluble) आंतों के अच्छे बैक्टीरिया द्वारा किण्वित होकर शॉर्ट चेन फैटी एसिड्स (SCFAs) जैसे ब्यूटिरेट बनाता है। ये SCFAs इंसुलिन सिग्नलिंग सुधारते हैं और लिवर में ग्लूकोज उत्पादन रोकते हैं। बाजरा, ज्वार, रागी जैसी अनाज रोज 30-50 ग्राम फाइबर देते हैं। भारतीय थाली के सुपरफूड्स गट के लिए बाजरा-ज्वार रोटी: हाई फाइबर, कम GI; बैक्टीरिया को फीड करते हुए शुगर स्पाइक रोकते हैं। दालें और छोले: प्रीबायोटिक फाइबर से Bifidobacteria बढ़ाते हैं, इंसुलिन संवेदनशीलता 20% तक बेहतर। हरी सब्जियां (पालक, भिंडी): इनुलिन जैसे फाइबर leaky gut ठीक करते हैं। दही/अचार: प्रोबायोटिक्स के साथ फाइबर मिलकर माइक्रोबायोम डाइवर्सिटी बढ़ाते हैं। रोजमर्रा के टिप्स: थाली से शुगर कंट्रोल सुबह बाजरा खिचड़ी, दोपहर दाल-सब्जी, रात रागी रोटी लें। 25-35 ग्राम फाइबर लक्ष्य रखें, पानी ज्यादा पिएं। 4 हफ्तों में स्टूल टेस्ट से माइक्रोबायोम सुधार दिखेगा, HbA1c घटेगा। व्हाइट राइस-मैदा छोड़ें। अध्ययन और भारतीय संदर्भ हाल के रिसर्च में पाया गया कि फाइबर-रिच भारतीय डाइट से डिस्बायोसिस 40% कम होता है, खासकर शहरी डायबिटीज मरीजों में। देसी खाना न सिर्फ सस्ता है, बल्कि गट हेल्थ के लिए वैज्ञानिक रूप से सिद्ध। https://pmc.ncbi.nlm.nih.gov/articles/PMC12332526/ https://www.lalpathlabs.com/blog/what-are-the-role-of-gut-health-in-diabetes-management/ https://hsph.harvard.edu/news/study-links-gut-microbiome-changes-to-increased-risk-of-type-2-diabetes/





