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Low Carb Sugar-Free Sweets & Cakes
Artinci was born out of Aarti's and Sumit's (Artinci's founders) abiding love for great-tasting dessert, while helping them stay committed to their health goals as well. As a result, Artinci makes delicious desserts with zero sugar, that are science and evidence-backed.
Aarti and Sumit come from a family of three generations of diabetics. They were themselves diagnosed pre-diabetic in 2012, and right there began a lifelong quest of a healthy, active lifestyle, including healthy swaps in food
Sugar free Sweets & Cakes
Aarti Laxman (Founder)
Artinci is founded by Aarti Laxman, a certified Metabolic coach in the Low-Carb Nutrition & Metabolic Health domain from dLife.in, India’s only legally tenable course in this subject—recognized by the NSDC (under the Ministry of Skill Development & Entrepreneurship, Govt. of India). It’s also internationally accredited by the CPD Standards Office UK, with a global record of 144 CPD hours—the highest for any course of its kind. The accreditation is both nationally valid and globally recognised in over 50+ countries..
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All about Sugar and sugar-free
The gut-brain axis connects your intestinal microbiome to brain centers controlling hunger and reward, influencing sugar cravings in diabetes through hormones like GLP-1 and microbial signals that modulate dopamine and appetite. Dysbiosis disrupts this axis, amplifying sweet cravings via inflammation and altered short-chain fatty acids (SCFAs), but restoring beneficial bacteria can boost GLP-1 secretion to curb overeating and stabilize blood sugar. Gut-Brain Signals and Cravings Gut microbes communicate with the brain via the vagus nerve, producing metabolites like SCFAs that stimulate GLP-1 release from intestinal L-cells, signaling fullness and reducing reward-driven sugar intake. In T2D, low diversity depletes Akkermansia muciniphila and Bifidobacterium, which normally enhance GLP-1 and curb ghrelin; this leads to heightened hypothalamic cravings and dopamine spikes from sweets, mimicking addiction. GLP-1: Microbiome's Natural Ozempic Beneficial bacteria ferment fiber into butyrate and propionate, activating GPR41/43 receptors to increase GLP-1, which slows gastric emptying, lowers post-meal glucose spikes, and dampens sweet cravings by acting on brain satiety centers. Studies link higher Faecalibacterium prausnitzii levels to elevated GLP-1 and fewer cravings in diabetics; prebiotics like FOS amplify this effect, mimicking GLP-1 agonists without drugs. Dysbiosis Fuels Sugar Addiction Harmful overgrowth of Proteobacteria and low SCFA producers inflame the gut-brain axis, raising LPS endotoxins that boost brain inflammation, ghrelin, and dopamine responses to sugar—creating a cycle of cravings and insulin resistancee. T2D patients show altered Firmicutes/Bacteroidetes ratios tied to compulsive eating; processed diets worsen this, but fermented foods restore balance. Indian Fixes for Gut-Brain Balance Start with daily dahi or chaach (probiotic-rich) paired with prebiotic garlic-onion tadka and millets to feed GLP-1-boosting bacteria; add methi or banana for FOS. Practice pranayama post-meal to calm vagus nerve signals, and swap late sweets for fiber-fermented kanji; track cravings via CGM for 2 weeks to see drops. https://www.dexcom.com/en-ca/blog/role-of-gut-health-in-blood-sugar-management https://pmc.ncbi.nlm.nih.gov/articles/PMC10698456/ https://www.eastsidefamilyhealth.com/blog/the-impact-of-gut-microbiota-on-diabetes-management-emerging-research-and-therapeutic-insights https://www.the-scientist.com/a-question-of-balance-how-the-gut-microbiome-influences-diabetes-70525
Gut Microbiome and Type 2 Diabetes: How ‘Good’ and ‘Bad’ Bacteria Change Your Sugar Levels
The gut microbiome, a vast community of trillions of bacteria in your intestines, plays a pivotal role in type 2 diabetes (T2D) by influencing insulin sensitivity, inflammation, and blood sugar regulation through dysbiosis—an imbalance where harmful bacteria outnumber beneficial ones. Loss of microbial diversity reduces protective short-chain fatty acids (SCFAs) like butyrate, worsening insulin resistance, while overgrowth of opportunistic pathogens triggers chronic low-grade inflammation that spikes glucose levels. What is Dysbiosis in T2D? Dysbiosis in T2D features reduced overall bacterial diversity, with a drop in Firmicutes/Bacteroidetes ratio and enrichment of pro-inflammatory groups like Proteobacteria and Betaproteobacteria. Studies across diverse populations show T2D patients have higher levels of pathogens such as Clostridium hathewayi, Clostridium symbiosum, and Escherichia coli, which promote leaky gut and endotoxemia, allowing bacterial toxins to enter the bloodstream and impair insulin signaling. This imbalance often precedes T2D, as seen in large cohort studies linking early microbiome shifts to elevated diabetes risk. Good Bacteria: Butyrate Producers and Protectors Beneficial bacteria like Faecalibacterium prausnitzii, Roseburia, and Bifidobacterium produce butyrate, an SCFA that strengthens the gut barrier, reduces inflammation via GLP-2 secretion, and enhances insulin sensitivity in liver and muscle cells. These species ferment dietary fiber into SCFAs, which activate receptors like GPR43 to lower fasting blood glucose and HbA1c; their depletion in T2D correlates with poorer glucose control. Restoring them through prebiotics or FMT shows promise in animal models for reversing insulin resistance. Bad Bacteria: Inflammation Drivers in T2D Harmful species such as certain Lactobacillus strains (in excess), Akkermansia muciniphila deficits, and opportunistic pathogens like Escherichia coli thrive in T2D, producing lipopolysaccharides (LPS) that fuel systemic inflammation and endothelial dysfunction. Elevated Betaproteobacteria and Bacilli link to advanced T2D stages, disrupting SCFA production and promoting metabolic endotoxemia, which exacerbates hyperglycemia and complications like fatty liver. Diet high in processed foods amplifies this shift, reducing diversity further. Indian Diet Tips to Balance Your Gut Incorporate fiber-rich Indian staples like dahi (probiotic source), garlic-onion tadka (prebiotic FOS), millets, and fermented idli/dosa to boost butyrate producers and crowd out pathogens. Aim for 25-30g daily fiber from dalia, bananas, and methi; pair with walking/yoga to enhance microbiome diversity and insulin response. Avoid late-night eating and ultra-processed maida items, which favor dysbiosis; monitor via CGM for personalized tweaks. https://pmc.ncbi.nlm.nih.gov/articles/PMC10698456/ https://www.frontiersin.org/journals/cellular-and-infection-microbiology/articles/10.3389/fcimb.2022.834485/full
प्रोबायोटिक्स का मधुमेह पर प्रभाव: क्या कहते हैं वैज्ञानिक अध्ययन?
प्रोबायोटिक्स क्या हैं और मधुमेह से कैसे जुड़े? प्रोबायोटिक्स जीवित अच्छे बैक्टीरिया हैं जो आंत के माइक्रोबायोम को संतुलित करते हैं। मधुमेह रोगियों में खराब आंत स्वास्थ्य इंसुलिन रेसिस्टेंस बढ़ाता है, जबकि प्रोबायोटिक्स सूजन कम कर रक्त शर्करा नियंत्रण में सहायक सिद्ध हुए हैं। भारत जैसे देश में जहां 77 मिलियन से अधिक टाइप 2 मधुमेह के केस हैं, ये पारंपरिक खाद्य जैसे दही से आसानी से मिल जाते हैं। टाइप 2 मधुमेह पर रिसर्च के मुख्य निष्कर्ष मेटा-एनालिसिस दिखाते हैं कि प्रोबायोटिक्स उपचार से फास्टिंग ब्लड ग्लूकोज (FBG) 3-13 mg/dL और HbA1c 0.3-0.5% तक कम हो सकता है, खासकर 6-12 सप्ताह में मल्टी-स्ट्रेन फॉर्मूला से। लैक्टोबैसिलस और बिफिडोबैक्टीरियम जैसे स्ट्रेन इंसुलिन संवेदनशीलता बढ़ाते हैं और लिपिड प्रोफाइल सुधारते हैं। सिनबायोटिक्स (प्रोबायोटिक्स + प्रीबायोटिक्स) अकेले प्रोबायोटिक्स से बेहतर परिणाम देते हैं। टाइप 1 मधुमेह में प्रभाव टाइप 1 में अध्ययन कम हैं, लेकिन मल्टीस्पीशीज प्रोबायोटिक्स से HbA1c और सूजन में कमी देखी गई। बच्चों में छोटे HbA1c ड्रॉप (~0.25%) नोट हुए, लेकिन बड़े ट्रायल्स की जरूरत है। प्रोबायोटिक दही और सप्लीमेंट्स के फायदे प्रोबायोटिक दही से T2D में FBG, HbA1c और LDL कम होता है। भारतीय घरों में घर का दही या छाछ रोजाना लें: बिना चीनी का, नट्स के साथ। सप्लीमेंट्स डॉक्टर की सलाह से लें, खासकर इंसुलिन यूजर्स के लिए। भारतीय आहार में शामिल करने के टिप्स रोज 1 कटोरी प्लेन दही या लस्सी पिएं, जिसमें लाइव कल्चर हो। प्रीबायोटिक्स के लिए प्याज, लहसुन, केला जोड़ें। पोर्शन कंट्रोल रखें और ब्लड शुगर मॉनिटर करें। सावधानियां और सलाह प्रोबायोटिक्स सहायक हैं, मुख्य उपचार नहीं; डॉक्टर से सलाह लें। व्यक्तिगत रिस्पॉन्स चेक करें, खासकर खराब कंट्रोल वाले मामलों में। संतुलित डाइट, व्यायाम के साथ मिलाकर इस्तेमाल करें। https://pmc.ncbi.nlm.nih.gov/articles/PMC8166562/ https://tap.health/probiotics-prebiotics-diabetes/ https://www.frontiersin.org/journals/endocrinology/articles/10.3389/fendo.2024.1392306/full
What Research Says About Probiotics and Blood Sugar in Type 1 and Type 2 Diabetes
Probiotics in Type 2 Diabetes Multiple meta-analyses confirm probiotics reduce FBG by 3-13 mg/dL and HbA1c by about 0.3-0.5% in T2D patients, especially over 6-12 weeks with multi-strain formulas. Effects are more pronounced in non-insulin users and those with baseline FBG over 130 mg/dL, alongside improvements in HOMA-IR and lipids. Network meta-analyses rank high-dose, multi-strain probiotics highest for glycemic efficacy. Evidence for Type 1 Diabetes T1D trials are fewer; a multispecies probiotic study showed trends toward lower HbA1c and insulin needs after 12 weeks, without statistical significance. Synbiotics reduced HbA1c, FBG, and hs-CRP significantly in an 8-week T1D trial. Pediatric meta-analyses note small HbA1c drops (~0.25%) but call for larger studies. Probiotic Yogurt Findings Probiotic yogurt trials in T2D lowered FBG, HbA1c, LDL, and blood pressure versus plain yogurt, with benefits after 4 weeks. A meta-analysis of yogurt-specific RCTs supports modest glycemic improvements, tied to strains like Lactobacillus. Synbiotic Supplements Synbiotics (probiotics + prebiotics) further cut FBG by 14 mg/dL in metabolic syndrome and T1D/T2D, outperforming probiotics alone in some glycemic markers. Meta-analyses highlight time-dependent effects: 6-8 weeks for HbA1c/insulin reductions. Key Limitations and Advice Effects vary by strain, dose, duration, and baseline control; probiotics work best as adjuncts. Consult doctors before starting, especially with insulin; monitor personal responses. https://www.dovepress.com/the-beneficial-effects-of-a-multispecies-probiotic-supplement-on-glyca-peer-reviewed-fulltext-article-DMSO https://www.sciencedirect.com/science/article/abs/pii/S0899900718303411 https://pmc.ncbi.nlm.nih.gov/articles/PMC8166562/





